Conclusions
There exists a plethora of diseases affiliated with CM-I, many of which have been mentioned in this article and certainly more to be discovered in the future. While the final outcome of CM-I may be the same, the strength of the correlation and pathophysiological mechanisms of each differs greatly and some may be spurious associations. Thus, the need for additional genetic research and investigation of CM-I continues.
Disclosure
The authors report no conflict of interest concerning the materials or methods used in this study or the findings specified in this paper.
Author contributions to the study and manuscript preparation include the following. Conception and design: Tubbs, Oakes. Acquisition of data: Tubbs, Loukas, Shayota, Oelhafen. Analysis and interpretation of data: Tubbs, Miller, Oakes. Drafting the article: Tubbs, Loukas, Miller. Critically revising the article: Tubbs, Shayota, Oelhafen, Chern. Reviewed submitted version of manuscript: Tubbs, Loukas, Chern, Miller, Oakes. Approved the final version of the manuscript on behalf of all authors: Tubbs.
Neurosurg Focus. 2011;31(3):e3 © 2011 American Association of Neurological Surgeons
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